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polymérases virales sont associées à des protéines de l’hôtes lors de la réplication des rétroïdes (e.g. Hepatitis B hepadnavirus HBV-Hsp90 [Hu, 2006 #3516]), des virus à ARN infectant les animaux (e.g. influenza [Naito, 2007 #3515] ainsi que pour les virus à ARN infectant les plantes (e.g. Cucumber necrosis tombusvirus [Serva, 2006 # | polymérases virales sont associées à des protéines de l’hôtes lors de la réplication des rétroïdes (e.g. Hepatitis B hepadnavirus HBV-Hsp90 [Hu, 2006 #3516]), des virus à ARN infectant les animaux (e.g. influenza [Naito, 2007 #3515] ainsi que pour les virus à ARN infectant les plantes (e.g. Cucumber necrosis tombusvirus [Serva, 2006 # | ||
+ | |||
+ | ====== Gene expression modification ======= | ||
+ | Cui et al 2007 Plant Cell | ||
+ | |||
+ | **Induced Expression of CDKC and CYCT1 Genes by | ||
+ | CaMV Infection** | ||
+ | |||
+ | Because of their roles in CaMV infection, we analyzed the | ||
+ | expression of these CDKC and CYCT1 genes in CaMV-infected | ||
+ | plants. Fully expanded lower leaves of 4-week-old Arabidopsis | ||
+ | plants were mechanically inoculated with CaMV. As described | ||
+ | above, it takes ;10 to 13 d after inoculation to first detect | ||
+ | significant levels of CaMV viral DNA and observe symptom | ||
+ | development in upper systemically infected leaves (Figure 1). As | ||
+ | shown in Figure 6, the levels of transcripts for CDKC;1, CDKC; | ||
+ | CYCT1;4, and CYCT1;5 were quite constant during the first 10 | ||
+ | DAI but increased substantially at 14, 18, and 24 DAI. Neither | ||
+ | mock inoculation nor TMV-cg or CaLCuV inoculation induced | ||
+ | expression of the genes (Figure 6). Thus, the four genes were | ||
+ | induced specifically by CaMV infection. | ||
+ | |||
+ | ====== Virus-host-vector interactions ====== | ||
+ | Mauck, K. E., C. M. De Moraes, et al. 2010 " | ||
+ | |||
+ | Previous studies have shown that vector-borne pathogens can alter the phenotypes of their hosts and vectors in ways that influence the frequency and nature of interactions between them, with significant implications for the transmission and spread of disease. For insect-borne pathogens, host odors are particularly likely targets for manipulation, | ||